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Table 2 Prevalence of genus group present according to obesity status, diabetes and insulin resistance status for the whole cohort (n=109) at baseline sampling

From: The association between obesity and weight loss after bariatric surgery on the vaginal microbiota

  Lactobacillus dominant
n/N (%)
Gardnerella dominant
n/N (%)
High diversity
n/N (%)
Total
n/N (%)
Obesity status
 Non-obese (BMI <30.0 kg/m2) 41/67 (61.2) 16/67 (23.9) 10/67 (14.9) 67/67 (100)
 Obese (≥30.0 kg/m2) 12/42 (28.6) 12/42 (28.6) 18/42 (42.8) 42/42 (100)
 Total 53/109 (48.6) 28/109 (25.7) 28/109 (25.7) 109/109 (100)
p valuea     0.002
Diabetes status
 Non-diabetic 49/97 (50.5) 25/97 (25.8) 23/97 (23.7) 97/97 (100)
 Diabetic 4/12 (33.3) 3/12 (25.0) 5/12 (41.7) 12/12 (100)
 Total 53/109 (48.6) 28/109 (25.7) 28/109 (25.7) 109/109 (100)
P value     0.451
Insulin resistance statusb
 Non-insulin resistant 4/10 (40.0) 2/10 (20.0) 4/10 (40.0) 10/10 (100)
 Insulin resistant 2/7 (28.6) 2/7 (28.6) 3/7 (42.9) 7/7 (100)
 Total 6/17 (35.3) 4/17 (23.5) 7/17 (41.2) 17/17 (100)
p value     1.000
  1. BMI body mass index
  2. aA p value of less than 0.05 demonstrates a significant difference in the distribution of genus group present according to obesity, diabetic and insulin resistance status. Fisher’s exact test employed as small numbers were present for each group (e.g. diabetic)
  3. bWhere concomitant fasting serum samples were available, fasting glucose and fasting insulin levels were identified. Using these values, we were able to calculate the HOMA-IR, according to the formula: the product of fasting insulin (μU/L) multiplied by fasting glucose (nmol/L) divided by 22.5. The 2nd tertile was used as the cut-off to determine insulin resistance status. Insulin resistance cut-off value, 2.98