Fig. 4
From: Growth rate determines prokaryote-provirus network modulated by temperature and host genetic traits
Effects of genes on the negative GrSRs throughout the infection cycle, including the stages of adsorption, establishment, and viral release. The negative GrSRs were significantly (P < 0.05) strengthened by the presence of genes responsible for host viral receptors of flagellum and pili, temperature-dependent lytic switches, and phage anti-CRISPR systems but decoupled by host immune systems, including the CRISPR-Cas and RM systems. CRISPR-Cas system: clustered regularly interspaced short palindromic repeats arrays (CRISPR) and CRISPR-associated protein (Cas) system. RM system, restriction-modification system; HSP, heat shock protein; CSP, cold shock protein